In recent years, Brugada syndrome has increasingly assumed greater importance in the clinical setting given the high number of young people between 30 and 40 years of age who have died due to it. The cause of this syndrome is not entirely clear, but is attributed either to a rapid inactivation of sodium channels (a congenital hereditary cause) or to the presence of a structural abnormality in the right ventricle with the presence of a replacement fibro-adipose tissue. By studying the electrical activity of the heart it is possible to determine the presence of the syndrome in the patient and also to define the stage in which it is present.