Sleep disturbances are common in Parkinson’s disease (PD) and often emerge early, suggesting that sleep may be a potential modulator of the clinical manifestations of the disease. While levodopa effectively treats motor symptoms, high-dose use can cause motor fluctuations and levodopa-induced dyskinesias (LID). This study investigates whether dyskinetic (DYS) patients differ from non-DYS PD patients and healthy controls in terms of slow-wave activity and brain signal complexity - key markers of sleep homeostasis and neural information processing. The analysis focused on the deepest stage N3 of non-REM sleep, which is considered crucial for the restoration of synaptic balance according to the synaptic homeostasis hypothesis.