Myocardial infarction is the primary cause of cardiac fibrosis, which often evolves into a condition known as heart failure. Alternative treatments to heart transplantation for late-stage heart failure are under investigation. In compliance with the 3Rs principle (Replacement, Reduction, Refinement), in-vitro devices are of a great interest since they replicate the behavior of cardiac tissue and reduce the employment of animal testing. In this thesis, a previously designed stretchable scaffold was characterized to prove its suitability for the in vitro engineering of human cardiac fibrotic tissue. A one-dimensional finite element method (FEM) model was developed and validated by an analytical model for different scaffold thicknesses.